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Our members asked: Are people with kidney disease at risk of getting too much potassium if they take a multivitamin? Which multivitamins are best for people with kidney disease?
Vitamin For Healthy Kidney Function
Because the kidneys play an important role in removing excess potassium from the blood, people with chronic kidney disease may be at risk of getting too much potassium, leading to excess blood levels. The level of risk depends on certain factors, such as the severity of the kidney disease, other comorbidities, and the use of medications that affect potassium levels (eg, diuretics, angiotensin receptor blockers [ARBs], and angiotensin-converting enzyme inhibitors [ACEIs]). ). (Diary Reference Intakes, National Academies Press 2019).
Multivitamins For People With Kidney Disease Who Are Restricting Potassium Intake
Some patients with chronic kidney disease may be advised by their doctor to limit their intake of potassium-rich foods, but multivitamins may not significantly increase potassium intake. That’s because most multivitamins are like that
Provide plenty of potassium, as most people are deficient in potassium and it is a mineral that is easily obtained from many types of food.
To contribute to your potassium intake, see the “Magnesium/Potassium” column in our Multivitamin Review Results table and choose a potassium-free product among the multivitamins listed as approved. If you have severe or advanced chronic kidney disease, you may have difficulty regulating your magnesium levels and should avoid magnesium supplements. Discuss your additional needs with your doctor.
If you are trying to avoid potassium, avoid “lithium” salts and salt substitutes and coconut water, as these products can cause potassium overload.
Are Cranberries Good For Kidneys?
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Kidney Support Supplements Improve Function Of Kidneys, Spleen & Gallbladder
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Author: Federica Belloni 1, †, Maria Kinquegran 1, †, Ramona Nicotera 2, Nazareno Carullo 3, Alessandro Casarella 3, Pierangela Presta 3, Michele Andreucci 3, Giovanni Squadrito 3, Giovanni Squadrito 3, Giovanni Squadrito 3, , †, Ramona Nicotera, 2, De Saro 3, Davide Bolgnano 3 and Giuseppe Coppolino 3, *
Foods That Are Good For The Kidneys
Received: 29 March 2022 / Revised: 7 May 2022 / Accepted: 7 May 2022 / Published: 9 May 2022
Chronic kidney disease (CKD) is commonly associated with vitamin K deficiency. Some serious complications of CKD include cardiovascular disease (CVD) and skeletal fragility, with increased risk of morbidity and mortality. There is a complex pathogenetic relationship between hormonal and ionic disturbances, bone tissue and metabolic changes, and vascular calcification (VC) and is defined as chronic kidney disease-mineral and bone disorder (CKD-MBD). Poor vitamin K status appears to play an important role in the progression of CKD, as well as in the initiation and progression of bone and cardiovascular complications. Three types of vitamin K are currently known: vitamin K1 (phylloquinone), vitamin K2 (menaquinone) and vitamin K3 (menadione). Vitamin K plays a variety of roles, including activation of vitamin K-dependent proteins (VKDPs) and modulation of bone metabolism, and contributes to the inhibition of VC. This review focuses on the biochemical and functional characteristics of vitamin K, suggests this nutrient as a potential marker of renal, CV, and bone damage in the CKD population, and explores its potential use for health promotion in this clinical setting. Treatment strategies for CKD-related osteoporosis and CV disease should include vitamin K supplementation. However, further randomized clinical trials are needed to evaluate the safety and adequate dosage for the prevention of these CKD complications.
Chronic kidney disease (CKD) is characterized by simultaneous vascular calcification and bone metabolism disorders [1]. In particular, an imbalance of the bone-vascular axis has been shown, which causes changes in both blood vessels and bones [2]. Although the mechanistic relationship of this cross-talk between the vascular and skeletal systems is not yet well understood, certain hormones, including parathyroid hormone (PTH) and 1, 25-dihydroxyvitamin D3, regulate skeletal and vascular mineralization. Regeneration of stem cells [3]. Therefore, the term “calcification paradox” was coined to link ectopic mineralization in blood vessels with decreased bone mineral density (BMD) [4]. In recent years, knowledge of the essential role of vitamin K has grown exponentially as it is well recognized in vascular calcification, cardiovascular disease, and bone destruction. Recently, increasing evidence suggests that vitamin K supplementation may be instrumental in preventing the rapid progression of vascular calcification and maintaining bone health in patients with CKD [5]. In this context, we aimed to focus on the biological functions of vitamin K, its relationship between cardiovascular disease (especially in hypertensive patients) and bone metabolism in CKD, and the potential use of vitamin K for health promotion. In this clinical setting.
A comprehensive review of the existing literature was conducted. First, studies were retrieved from the PubMed, Scopus, and Web of Knowledge online databases using the following keywords: “chronic kidney disease,” “vitamin K,” “vascular calcification,” “bone metabolism,” “osteoporosis,” and “heart.” diseases”. Online searches were pre-filtered by language (English) and availability of full-text articles. In addition, reference lists of included studies were reviewed to identify potentially relevant studies missed during the database search. Online searches will end forever on March 15, 2022.
Vitamin D & B Complex For Kidney Function — Thriving With Ckd
The term vitamin K, or naphthoquinone, refers to a family of water-soluble molecules with a similar structure, but different origin and function, formed by the 2-methyl-1, 4-naphthoquinone ring. Three main forms are currently known, defined as vitamers, which differ in their side chains linked to the 2-methyl-1, 4-naphthoquinone ring at position 3 [6]; Specifically, these are vitamin K1 (phylloquinone), vitamin K2 (menaquinone) and vitamin K3 (menadione). The main known biological function of vitamin K1 is blood coagulation, as it acts as a cofactor for the enzymatic conversion of glutamic acid (Glu) residues in vitamin K-dependent proteins (VKDPs) to gamma-carboxyglutamic acid (Gla). Vitamin K-dependent gamma-glutamyl carboxylase is localized in the endoplasmic reticulum of all mammalian tissue cells [7, 8, 9] and is necessary for the conversion of protein-bound glutamate to carboxy-glutamate, which is necessary for II, VII, IX. and X for factors of the coagulation cascade and natural anticoagulants proteins S and C [10, 11]. Sources of vitamin K1 are mainly leafy or flowering vegetables (spinach, lettuce, broccoli, cabbage, Brussels sprouts, turnips), but chickpeas, beans, soybeans, green tea, eggs, pork and beef liver also contain vitamin K1. [12]. Vitamin K2 is mainly synthesized by the gut microbiota and is designated as menaquinone (MC); Several different forms can be identified, numbered from 4 to 13, depending on the length of the isoprene chain attached to the methylated naphthoquinone ring. MK-4 is derived from the conversion of phylloquinone or menadione and is mainly found in meat and animal products. – Products such as eggs, cow’s milk and yogurt [13, 14, 15]. MK-7, on the other hand, is a long-chain type produced by gut bacteria and is found in fermented foods such as cheese and soy [16]. MK4 and MK7 are the two most common menaquinones in the human diet, along with MK8, MK9, and MK10 [13]. Vitamin K3, also known as menadione, was previously considered to be a synthetic form of vitamin K. However, it has been shown that vitamin K3 can originate in the intestines.