Lose Weight Tips In Urdu – The GM Diet Plan was created for General Motors employees, hence the name GM Diet. The diet plan promises to help a person lose five to seven kilos in seven days. For seven days, the diet recommends eating foods from different food groups. This diet promises more weight loss in less time than any other diet.
The GM diet plan not only helps in weight loss but also has other health benefits including improved digestion, detoxification of the body and improved ability of the body to burn fat later.
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It has been proven helpful for most people following a GM diet. This is because the diet includes vegetables and fruits that are low in calories. There are even negative calorie foods in the diet, which take you to digest more calories than they give you. The diet contains a lot of water and therefore helps in detoxification of the body. The diet can be followed several times, but with a gap of five to seven days between them.
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Day 1: Any fruit except banana. You can eat fruit as often as you want. Watermelons are recommended for easy weight loss as they contain a lot of water.
Day 2: You can eat any vegetables, cooked or raw, as often as you want. However, eat a medium-sized potato for breakfast with a teaspoon of low-fat butter.
Day 3: Apart from bananas and potatoes, you can eat cooked/raw fruits and vegetables throughout the day.
Day 4: Eat only bananas and milk on day 4. It’s eight large bananas with three glasses of skimmed milk.
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Day 5: Chicken/fish with six whole tomatoes (280g). Vegetarians can have brown rice or cottage cheese instead of meat. Increase your water intake.
Day 6: Unlimited vegetables (preferably spinach) but no potatoes. Vegetarians can have brown rice or cottage cheese, non-vegetarians can have chicken/fish (280 g). Have plenty of water.
Walk for 45 minutes on these seven days. If you feel weak, avoid strenuous exercise routines.
Beans, sweets and high calorie drinks should be avoided. You can have black tea/herbal tea or black coffee without adding any sweetener.
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Once you complete one week of the GM diet plan, it is recommended that you eat high protein and low carbohydrates the following week. Avoid junk as it will add your weight again.
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Learn about Lifestyle, Fashion & Beauty Trends, Relationship Tips, Health & Food Jolla- Researchers have developed a new type of pill that tricks the body into thinking it has eaten calories and instead burns fat. The compound effectively prevented weight gain, lowered cholesterol, controlled blood sugar and reduced inflammation in mice, making it an excellent candidate for rapid transition to human clinical trials.
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Unlike most diet pills on the market, this new pill, called fexaramine, doesn’t dissolve in the bloodstream like appetite suppressants or caffeine-based diet pills, but stays in the gut with fewer side effects.
Ronald Evans, director of the Gene Expression Laboratory, has developed a compound called fexaramine that acts as a hypothetical food. Fexaramine, which tricks the body into responding as if it has consumed calories, may lead to effective treatments for obesity and diabetes in humans.
“This pill is like a hypothetical food,” says Ronald Evans, director of the Gene Expression Laboratory and senior author of the new paper, published Jan. 5, 2015, in Nature Medicine. “It sends out the same signals that normally happen when you eat a lot of food, so the body starts clearing space to store it. But no change in calories and appetite.
In the United States, one-third of adults are obese and 29.1 million people have diabetes, according to the Centers for Disease Control and Prevention. Obesity and diabetes contribute to increased health costs, increased risk of health complications, and reduced life expectancy.
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Researchers at the institute have developed a fat-burning compound called fexaramine, which causes weight loss in animal models without the usual side effects. Fexeramine causes the body to respond as if calories have been consumed and may lead to effective treatment of type 2 diabetes and obesity in humans.
Evans’ lab has spent nearly two decades studying the farnesoid X receptor (FXR), a protein that controls how the body releases bile from the liver, digests food, and stores fat and sugar. The human body turns on FXR at the start of a meal, preparing for food flow, Evans et al. FXR not only stimulates the release of bile acids for digestion, but also alters blood sugar levels and causes the body to burn some fat in preparation for the incoming meal.
Pharmaceutical companies that aim to treat obesity, diabetes, liver disease, and other metabolic conditions have developed systemic drugs that activate FXR, turning on several pathways regulated by FXR. But these drugs affect many organs and cause side effects. Evans wondered if altering FXR only in the gut, as opposed to the gut, liver, kidneys, and adrenal glands, would have a different effect.
Researchers have shown that fexaramine stops weight gain and fat burning in animal models. Fexaramine is absorbed in the intestine and does not enter the bloodstream, so it does not cause the side effects of ordinary diet pills. After further testing, researchers believe this could lead to an effective weight loss diabetes treatment for humans.
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“When you eat, you have to quickly activate a series of reactions throughout the body,” says Evans. “The truth is that the gut is the first responder to all of this.”
Evans and his colleagues developed the fexeramine compound in a departure from the drug scaffolding used by most pharmaceutical companies to target FXR. “When we give it orally, it only works in the gut,” explains senior staff scientist Michael Downs, an author of the new work. If such a drug is given daily in pill form that only reaches the gut — not carried throughout the body into the bloodstream carrying the drug — not only would the side effects be prevented, but so would the compound’s success in preventing weight gain.
When obese mice were given fexaramine for five weeks, the mice lost weight, lost fat and had lower blood sugar and cholesterol levels than untreated mice. In addition, the mice had an increase in body temperature — indicating a higher metabolism — and some of the white fat deposits in their bodies turned into beige forms of healthy, energy-burning tissue. Even the collection of bacteria in the mice’s guts changed when they received the drug, although it’s not yet clear what those changes mean.
Why does fexaramine work better in the gut than drugs that simultaneously activate FXR throughout the body? Evans thinks it has to do with the natural order in which the body’s molecular pathways normally respond to food.
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“The body’s response to food is like a relay race, and if you tell all the runners to go at the same time, you’ll never pass the baton,” says Evans. “We have learned how to motivate the first runner so that the rest of the events occur in a natural sequence.”
Because fexaramine does not reach the bloodstream, it is safer in humans than other FXR-targeting drugs, the researchers hypothesized. They are already working to establish human clinical trials to test fexaramine’s effectiveness in treating obesity and metabolic diseases. Medication given under a doctor’s prescription may work in conjunction with diet and lifestyle changes, such as weight loss surgery or other obesity or diabetes medications.
Other researchers on the study include the institute’s Sunsoon Fang, Jay Myung Su, Elizabeth Yu, Eiji Yoshihara, Sandra Jacinto, Yelizaveta Lukasheva, Annette Atkins, and Ruth Yu; Shannon Reilly and Alan Saltiel from the University of Michigan; Olivia Osborne, Denis Lackey, Bernd Schnable, David Brenner, and Jerrold Olefsky from the University of California, San Diego; Alessia Perrino and Christina Schoonjans from Ecole Polytechnique Fédérale de Lausanne; Alexander Khwat of Chemdiv; Sally Coulter and Christopher Liddle from the University of Sydney.
National Institutes of Health, Glenn Foundation for Medical Research, Leona M. and Harry B. This work was supported by grants from the Helmsley Charitable Trust, Ipsen Bioscience, the California Institute for Regenerative Medicine, the Ellison Medical Foundation, and National Health. and medical research. Australia Council. Ronald Evans also receives funding from the Howard Hughes Medical Institute.
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Sungsoon Fang, Jae Myong Su, Elizabeth Yu, Eiji Yoshihara, Sandra Jacinto, Yelisaveta